Κουπί Ποικιλία περισκόπιο ellen nollen google scholar Πρεσβευτής Διατεθειμένος ιδιοσυγκρασία
The therapeutic potential of targeting tryptophan catabolism in cancer | British Journal of Cancer
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Alliance for Healthy Aging
Ellen Nollen | VIB Conferences
Tryptophan metabolism: entering the field of aging and age-related pathologies: Trends in Molecular Medicine
Crystal Structure and Mechanism of Tryptophan 2,3-Dioxygenase, a Heme Enzyme Involved in Tryptophan Catabolism and in Quinolinate Biosynthesis, | Biochemistry
Frontiers | Regulation of Age-Related Protein Toxicity
Frontiers | Regulation of Age-Related Protein Toxicity
Proteome-wide Changes in Protein Turnover Rates in C. elegans Models of Longevity and Age-Related Disease - ScienceDirect
Frontiers | Squalamine and Its Derivatives Modulate the Aggregation of Amyloid-β and α-Synuclein and Suppress the Toxicity of Their Oligomers
Cornelis CALKHOVEN | University of Groningen, Groningen | RUG | European Research Institute for the Biology of Ageing | Research profile
Protein quality control: From mechanisms to disease
Frontiers | Key Metabolic Pathways in MSC-Mediated Immunomodulation: Implications for the Prophylaxis and Treatment of Graft Versus Host Disease
Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity: Cell
Probing the Origin of the Toxicity of Oligomeric Aggregates of α-Synuclein with Antibodies | ACS Chemical Biology
Tobacco carcinogen induces tryptophan metabolism and immune suppression via induction of indoleamine 2,3-dioxygenase 1 | Signal Transduction and Targeted Therapy
Speakers | ARDD 2022
Pawanrat CHALORAK | Instructor | PhD | Chulalongkorn University, Bangkok | Department of Radiological Technology | Research profile
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Julius Bier Kirkegaard — Theoretical Biophysics & Data Science
A Brain-Permeable Aminosterol Regulates Cell Membranes to Mitigate the Toxicity of Diverse Pore-Forming Agents | ACS Chemical Neuroscience
Delivery of Native Proteins into C. elegans Using a Transduction Protocol Based on Lipid Vesicles | Scientific Reports
Deletion of SERF2 in mice delays embryonic development and alters amyloid deposit structure in the brain | Life Science Alliance
Probiotic Bacillus subtilis Protects against α-Synuclein Aggregation in C. elegans - ScienceDirect
Akira KITAMURA | Senior Lecturer | Ph.D. | Hokkaido University, Sapporo | Hokudai | Faculty of Advanced Life Science | Research profile
Cognitive impairments correlate with increased central nervous system immune activation after allogeneic haematopoietic stem cell transplantation | Leukemia
Deletion of SERF2 in mice delays embryonic development and alters amyloid deposit structure in the brain | Life Science Alliance
MOAG-4 promotes the aggregation of α-synuclein by competing with self-protective electrostatic interactions - ScienceDirect